Author name: Don Gerig, RDCS

In part 1 of our hypertrophic cardiomyopathy (HCM) series, we reviewed the key echocardiographic clues that should raise suspicion for hypertrophic cardiomyopathy. In part 2, we focused on how to differentiate LVOT obstruction flow from mitral regurgitation. Now, in this third and final article, we’ll cover one of the most important ways to unmask obstruction

In part one of our HCM series you learned key echocardiographic clues that should raise suspicion for hypertrophic cardiomyopathy.  In this article we’ll look at one of the more challenging aspects of scanning patients with obstructive hypertrophic cardiomyopathy: accurately differentiating LVOT vs Mitral Regurgitation in HCM. Both produce high-velocity systolic jets and sometimes even overlap

CardiServ is committed to supporting individuals and organizations that share our values of inspiring excellence in echocardiography.  I recently had the opportunity to interview Michael Owen, RDCS, RVT, a sonographer and the developer of EchoWorkflows.com. I also had the chance to put his tool to the test in a real-world clinical environment in a busy

Both constrictive pericarditis and restrictive cardiomyopathy limit ventricular filling, but for completely different reasons.

They share nearly identical symptoms and often similar Doppler findings such as dyspnea, edema, JVD, preserved EF and abnormal filling, which is why they’re so commonly mistaken for one another.

But they arise from completely different mechanics, one from the outside of the heart, one from the inside.

Learn how to diagnose cardiac tamponade using echocardiographic signs, Doppler clues, and hemodynamic changes. Identify critical pressure patterns before clinical shock develops.

After learning the pericardial disease spectrum in Part 1 of our pericardial disease series, the natural next step is mastering the first question in the Three-Step Framework: Is there fluid? Seems like a simple enough thing to do, right? Well, it’s not always as straightforward as you might hope.  Misinterpreting an effusion can lead to

While we often think of the pericardium as a protective sac, it’s so much more than that. Under normal conditions, it moves and stretches with every heartbeat, quietly accommodating changes in volume and pressure. 

But when it loses that flexibility, the pericardium can transmit pressure back to the heart or even restrict its ability to fill. 

And that’s where the pericardial spectrum begins. A continuum that ranges from simple effusion to hemodynamic tamponade and, in chronic or recurrent inflammation, to constriction. Understanding this progression changes the way we view the pericardium and how we interpret our echo findings.